Looks like there is a known mechanism behind nerve damage from EMF from the 60's.
See below. I wonder what we can do about it?.. Seems an enzyme gets inhibited, .. how to un-inhibit, or how to create more of it.. no idea! It's acting like a pesticide! It's also interesting that these levels can be tested, but some say the testing is not accurate unfortunately. https://ecfsapi.fcc.gov/file/1954790005.pdf They say: " Soviet research of electromagnetic radiation health effects in the 1960's and 1970's found that nonthermal EMF exposures could produce effects similar to other toxins, such as chemicals and heavy metals.20 One consistent finding from the Soviet research was that when humans and animals were exposed to radio wave or microwave frequency, this radiation would inhibit cholinesterase, producing an excess of acetylcholine - the same effect as organophosphate pesticides. 5,21,22 Some Soviet EMF researchers specifically pinpointed this enzyme effect as a significant factor leading to nervous system disfunction. 5,20,23 (Phase one of microwave sickness sometimes produced an increase in recorded cholinesterase, prior to phase two which evidenced decreased cholinesterase activity and central nervous system disfunction.)21" "In a 1973 Soviet study, blood cholinesterase reduced to below 70% of pre-exposure levels in rats and rabbits exposed to the 30-300 MHz range at intensities of only .01 mW/cm2or .0019mW/cm2for 4 months. 23 According to the U.S. National Institute of Occupational Safety and Health (NIOSH), when organophosphate pesticide exposure results in a decrease of blood cholinesterase to less than 70% of the pre-exposure level, that exposure is considered unacceptable due to the health hazard. 13 A 1994 Soviet study by the Russian Institute of Cell Biophysics produced an average drop in brain cholinesterase to 70% of pre-exposure levels in rats after one 60 minute exposure to pulsed 880 MHz at 1.5mW/cm2. 24 Also, a U.S. government study conducted in cooperation with Soviet researchers in an effort to replicate Soviet findings discovered that microwaves of 2.45 GHz at .5mW/cm2 intensity resulted in blood cholinesterase inhibition in rats exposed during three months for seven hours daily. 6 This study confirmed prior Soviet accounts of nonthermal microwave health effects. " The US level permitted is 1mW/cm2 , Russians Chinese is 0.01mW/cm2. Canada is 0.43mW/cm2 The nervous system seems to be targeted because of the cholinesterase reduction. Also references in there that effects seems to be cumulative, found by the Russians. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5897318/ The results revealed that WiFi exposure caused a significant increase in anxiety level and affect locomotor function. Furthermore, there was a significant decrease in AChE activity with a concomitant increase in AChE mRNA expression level in WiFi exposed rats when compared with control. In conclusions, these data showed that long term exposure to WiFi may lead to adverse effects such as neurodegenerative diseases as observed by a significant alteration on AChE gene expression and some neurobehavioral parameters associated with brain damage. |
Ok so there is one thing that might help: Dilantin and atropin.
Magnesium salts was also found to help. The importance of this enzyme effect in producing ES-type symptoms from chemicals and from chronic, low-level EMF exposures leads to a mechanism of action for explaining many EMF-related health effects by using conventional medical and scientific knowledge of poisons. That radiation exposure at nonthermal, nonionizing doses can produce the same effect as chemical poisons such as organophosphate pesticides provides sound evidence for a radiation poisoning effect. It also may explain why some MCS patients become ES too and why some ES also become MCS. The agents are different (chemical vs. physical), but the effect is the same - inhibiting cholinesterase. When trying to conceptualize a problem, it is often helpful to know what the opposite of the problem might be. In this case, the disease myasthenia gravis (MG) appears to have characteristics the opposite of organophosphate pesticide poisoning and cholinesterase inhibition. MG patients may take a type of medication called anticholinesterase, which purposely reduces cholinesterase in order to increase acetylcholine.25 MG is generally considered an autoimmune disease of muscle weakness diagnosed by a blood test for antibodies to the acetylcholine receptor. 25 Drugs that make MG worse include magnesium salts and the prescription drugs Dilantin and atropine. 25. 26 In one Soviet study, Dilantin was shown to be beneficial in treating patients with hypothalamic syndrome, a phase of microwave sickness; magnesium, which assists enzyme processes, has been helpful for some ES. 19,27 Some animal studies with Dilantin have shown a reduction in acetylcholine due to the drug's effect.28 Dilantin also has shown a protective effect against incesticide poisoning, specifically regarding organophosphates and DDT.28 (However, Dilantin (phenytoin) appears on a medical list of drugs not recommended for those with porphyria.)29 Organophosphate pesticide poisoning is often treated with the prescription drug atropine or other drugs.18 Atropine was also used by the Soviets for treating some cases of microwave sickness. 30,31 Summarizing the clues here: • Atropine and Dilantin are linked to antidote or protective effects regarding organophosphate pesticide poisoning. • Atropine was used by the Soviets in treating some cases of microwave sickness. • Dilantin was reported to be helpful in treating hypothalamic syndrome, the final phase of microwave sickness. • Magnesium is helpful for some ES. Magnesium salts, Dilantin, and atropine can make myasthenia gravis patients worse-their illness appearing to have characteristics opposite of radiation poisoning and organophosphate poisoning which inhibit the enzyme cholinesterase. |
I'd say magnesium is probably the best of anything at helping the enzyme activity get better.. The others have not been proven to help.
I think this is a very important part of the mechanism of how EMFs damage nerves, and I'm surprised it's not talked about more often. |
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